Pain Dissolution
Many of us have the tendency to ignore or suppress the pain sensation until it cannot longer be ignored. Then we want immediate relief. The options are:
I/ Indirect Pain relief, practiced on you by physicians and therapists:
- Removing the Symptom through the absorption of Non-Natural Medications: Different pain medications act at different places in the pain pathways. The type depends upon the possible source of the pain, the level of discomfort, and the detrimental responses of the body to many synthetic medications.
- Non-opioid analgesics, like ibuprofen (Advil), aspirin, acetaminophen (Tylenol), and naproxen (Aleve), act at the site of pain. The damaged tissue releases enzymes that stimulate local pain receptors. Non-opioid analgesics interfere with the enzymes and reduce inflammation and pain. They can have some adverse effects in the liver and kidneys and can cause gastrointestinal discomfort and bleeding with prolonged use.
- Opioid analgesics act on synaptic transmission in various parts of the central nervous system by binding to natural opioid receptors. They inhibit ascending pathways of pain perception and activate descending pathways. Opioid analgesics are used for higher levels of pain relief. They include morphine, meripidine (Demerol), codeine, propoxyphene (Darvon), fentanyl, oxycodone. In overdose, they become addictive.
- Adjuvant analgesics (co-analgesics) are primarily used for treating other conditions, but they also relieve pain. They are useful in treating neuropathic pain (chronic pain that comes from injury to the CNS). They include the following:
- Anti-epileptic drugs. They reduce membrane excitability and action potential conduction in neurons of the CNS.
- Tricyclic anti-depressants. They affect synaptic transmission of serotonin and certain neurons in the CNS, thereby affecting pain-modulating pathways.
- Anesthetics block action by interfering with sodium and potassium channels in nerve cell membranes. Examples include lidocaine, novocaine and benzocaine.
